[HydrogenBond]

If we look at the cell, the lion's share of the cell useable energy or ATP goes into the pumping of cations out of the cell's membrane, i.e., sodium pumps. This energy creates a slight voltage potential across the cell membrane where the outside becomes positive due to the accumualtion of sodium cations outside the cell. The concentration gradient with the inside of the cell makes sodium want to get back into the cell causing sodium to collect on the outside surface of the membrane.

The effect of all these localized sodium cations is to compete with the hydrogen of the local exterior water for the electron density of the oxygen of water, i.e, hydration spheres of sodium. This creates a hydrogen bonding potential within the local hydrogen. With the outside sodium cations being a steady state arrangement due to the constant flux of ATP into the membrane, the hydrogen of water has no choice but to propogate its induced potential into the water outside the cell. This electrophilc potential or this slight need for electron density helps draws reduced food materials toward the cell. The more amplification within this H-bonding signal going beyond the cell, the farther out it can impact the environment. The membrane will cherry pick what it needs from this eternal buffet, through its distribution of transport proteins, and use the membrane potential to bring the snack into the cell.

When a cell goes into a cell cycle, the membrane potential lowers because more sodium is able to flow back into the cell, i.e.,, ion pump reversal. This lowers the H-bonding amplification outside the cell, helping to isolate the cell from the environment. Even though the voltage potential across the membrane drops during cell cycles the amount of ATP going into the sodium pumps increases. The cell cycle will lower the voltage but will increase the current going back and forth within the membrane. The net effect inside the cell are slightly higher H-bond signals (less negative), with much greater frequency. As a loose anology, if the water inside the cell was a pond, it will see bigger rocks and much more of them being thrown into the water. The result is that a stronger and more diversified h-bonding energy interference grid will be set up with the cell during cell cycles. Outside the cell has smaller rocks but many more of them.

When we reach very advanced cells like neurons, which can fire, what essentially is happening is that the H-proton amplication reaches a peak outside the neutron and then drops. Inside the cell will see the opposite or reflected effect. During outside peaks the inside is low and during outside lows the inside is at a peak. Outside the neuron the cyclic changes of potential will cycle the potential strength within the local water. During outside peaks the neuron is sending stronger H-bond signals far outwards toward lower potential. It can even send them to neutrons still powering up. During outside lows, the high average potential of the nervous environment causes the H-bonding potential of other neutrons to head toward that neuron. These can also come from neurons still powering up.

The hydrogen proton can move almost 100 times faster in water than any other ion. This is probably due to hydrogen not having a cumbersome hydration sphere. The implication is that hydrogen bond energy propagation within water will proceed cationic currents and maybe even chemical signals diffusing in the water. This is not only true outside the neutron, but the inside peak H signals within the inside water should reach the nuclear membrane and maybe the DNA long before any chemicals can get there. The H primes the pump, so to speak, by having a preliminary impact on the H-bonding potential environment within the neuron.